PHILADELPHIA: Researchers have released results from a study showing that a key protein associated with the development of Alzheimer’s disease, Amyloid precursor protein (APP), could potentially play an important role in eye and muscle health. The study, entitled “FE65 and FE65L1 amyloid precursor protein–binding protein compound null mice display adult-onset cataract and muscle weakness,” demonstrated that in a mouse model where proteins binding to APP, called FE65 and FE65L1, are deleted, they cause cataracts and muscle weakness. If the findings are confirmed in clinical studies, FE65 and FE65L1 proteins could be a future therapeutic target for cataracts, muscular dystrophy and Alzheimer’s disease.